This is a link to an article by an Indian media organization summarizing the report.
 http://www.infowars.com/2-billion-may-suffer-from-cell-phone-cancer-by-2020/
http://www.buergerwelle.de/pdf/cellphone_studies.pdf
Above is a link to a 76 page pdf. Below is the first segment. This is for those who claim there is no hard science to back up claims that RF in the frequeny range of cell phones, towers, wi fi and other gadgets will cause cancer and other diseases
15/7/03
Biological studies on radiation similar to that emitted by cell phones
Adey WR, Byus CV, Cain CD, Higgins RJ, Jones RA, Kean CJ, Kuster N,
MacMurray A, Stagg RB, Zimmerman G, Phillips JL, Haggren W,
Spontaneous and nitrosourea-induced primary tumors of the central
nervous system in Fischer 344 rats chronically exposed to 836 MHz
modulated microwaves. Radiat Res 152(3):293-302, 1999.
We have tested an 836.55 MHz field with North American Digital Cellular
(NADC) modulation in a 2-year animal bioassay that included fetal exposure. In
offspring of pregnant Fischer 344 rats, we tested both spontaneous
tumorigenicity and the incidence of induced central nervous system (CNS)
tumors after a single dose of the carcinogen ethylnitrosourea (ENU) in utero,
followed by intermittent digital-phone field exposure for 24 months. Far-field
exposures began on gestational day 19 and continued until weaning at age 21
days. Near-field exposures began at 35 days and continued for the next 22
months, 4 consecutive days weekly, 2 h/day. SAR levels simulated localized
peak brain exposures of a cell phone user. Of the 236 original rats, 182 (77%)
survived to the termination of the whole experiment and were sacrificed at age
709-712 days. The 54 rats (23%) that died during the study ("preterm rats")
formed a separate group for some statistical analyses. There was no evidence of
tumorigenic effects in the CNS from exposure to the TDMA field. However, some
evidence of tumor-inhibiting effects of TDMA exposure was apparent. Overall,
the TDMA field-exposed animals exhibited trends toward a reduced incidence of
spontaneous CNS tumors (P < 0. 16, two-tailed) and ENU-induced CNS tumors
(P < 0.16, two-tailed). In preterm rats, where primary neural tumors were
determined to be the cause of death, fields decreased the incidence of ENUinduced
tumors (P < 0.03, two-tailed). We discuss a possible approach to
evaluating with greater certainty the possible inhibitory effects of TDMA-field
exposure on tumorigenesis in the CNS.
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